- How much mag do I need for torsades?
- Is torsades VT or VF?
- Is polymorphic v tach the same as torsades?
- How do you identify Torsades de Pointes?
- Do you shock torsades?
- Do you have a pulse with torsades?
- Does a pacemaker prevent torsades?
- Can amiodarone cause torsades?
- What does torsades feel like?
- What can cause torsades?
- What are the lethal heart rhythms?
- Why does hypokalemia cause long QT?
- Is torsades de pointes hereditary?
- Is polymorphic VT the same as torsades?
- What are the 3 shockable rhythms?
- Can dehydration cause prolonged QT?
- Where does torsades de pointes originate?
- Does magnesium shorten QT interval?
- What is the drug of choice for torsades de pointes?
- How do you treat torsades?
How much mag do I need for torsades?
Magnesium can be given at 1-2 g IV initially in 30-60 seconds, which then can be repeated in 5-15 minutes.
Alternatively, a continuous infusion can be started at a rate of 3-10 mg/min..
Is torsades VT or VF?
Frequent PVCs with ‘R on T’ phenomenon trigger a run of polymorphic VT which subsequently begins to degenerate to VF. QT is difficult to see because of artefact but appears slightly prolonged (QTc ~480ms), making this likely to be TdP.
Is polymorphic v tach the same as torsades?
Polymorphic ventricular tachycardia This is commonly referred to as torsades de pointes, but it’s actually not the same thing. Polymorphic ventricular tachycardia may be caused by several etiologies (e.g. congenital QT prolongation, acquired QT prolongation, ischemia, Takotsubo’s cardiomyopathy).
How do you identify Torsades de Pointes?
Symptoms of torsades de pointes include:heart palpitations.dizziness.nausea.cold sweats.chest pain.shortness of breath.rapid pulse.low blood pressure.
Do you shock torsades?
Torsades de pointes is a ventricular tachycardia. In the unstable patient, cardiovert. In the pulseless, defibrillate. (The polymorphic nature of the rhythm may interfere with the defibrillator’s ability to synchronize, so cardioversion may not be possible.
Do you have a pulse with torsades?
Today one needs to be aware that drug-induced long QT syndrome is common and hence, a thorough medication history must be obtained. Patients with torsade may be hypotensive, have a rapid pulse and have loss of consciousness.
Does a pacemaker prevent torsades?
The pacemaker component of such devices should in theory help prevent torsades by preventing bradycardia. However, the rate of most pacemakers is not likely to provide protection from torsades.
Can amiodarone cause torsades?
4 Amiodarone is presumed to have a low incidence of drug-induced torsades de pointes (TdP) with an incidence of <0.5%.
What does torsades feel like?
You may suddenly feel your heart beating faster than normal, even when you’re at rest. In some TdP episodes, you may feel light-headed and faint. In the most serious cases, TdP can cause cardiac arrest or sudden cardiac death. It’s also possible have an episode (or more than one) that resolves quickly.
What can cause torsades?
Common causes for torsades de pointes include drug-induced QT prolongation and less often diarrhea, low serum magnesium, and low serum potassium or congenital long QT syndrome. It can be seen in malnourished individuals and chronic alcoholics, due to a deficiency in potassium and/or magnesium.
What are the lethal heart rhythms?
Ventricular fibrillation, ventricular tachycardia and prolonged pauses or asystole are dangerous. Arrhythmias associated with very low potassium or magnesium or those associated with inherited causes such as QT prolongation are also serious. The ones you mentioned are supra- ventricular and generally not lethal.
Why does hypokalemia cause long QT?
Hypokalemia is another common risk factor in drug-induced LQTS. Low extracellular potassium paradoxically reduces IKr by enhanced inactivation  or exaggerated competitive block by sodium . As a result, hypokalemia prolongs the QT interval.
Is torsades de pointes hereditary?
Genetic susceptibility is an important consideration in patients with drug-induced Torsades de Pointes (TdP) because it may be the sentinel event unmasking an underlying congenital long QT syndrome (LQTS). Previous studies have identified congenital LQTS in 5% to 20% of cases with drug-induced TdP.
Is polymorphic VT the same as torsades?
Polymorphic VT is defined as an unstable rhythm with a continuously varying QRS complex morphology in any recorded ECG lead. Polymorphic VT that occurs in the setting of QT prolongation is considered as a distinct arrhythmia, known as torsades de pointes.
What are the 3 shockable rhythms?
Shockable Rhythms: Ventricular Tachycardia, Ventricular Fibrillation, Supraventricular Tachycardia.
Can dehydration cause prolonged QT?
Electrolyte abnormalities Disturbances in the levels of ions such as sodium and potassium in your body due to conditions such as dehydration, severe diarrhea, and even eating disorders can cause long QT syndrome. The QT interval returns to normal when the ions get back to normal.
Where does torsades de pointes originate?
Conclusion The most frequent site of origin of TdP is the outflow tract. Further studies are needed to understand why this relatively small area of the ventricle is a predominant site of origin of diverse ventricular arrhythmias.
Does magnesium shorten QT interval?
Magnesium sulfate reduced the risk of an ibutilide- induced QTc interval increase of greater than 30 msec or greater than 60 msec and reduced the risk of a QTc interval value of more than 500 msec by 65%, 60%, and 68%, respectively (p=0.07, p=0.175, and p=0.160). Conclusions.
What is the drug of choice for torsades de pointes?
Treatment of torsade de pointes includes: isoproterenol infusion, cardiac pacing, and intravenous atropine. Intravenous magnesium sulfate, a relatively new mode of therapy for torsade de pointes, was proven to be extremely effective and is now regarded as the treatment of choice for this arrhythmia.
How do you treat torsades?
Treatment is with IV magnesium, measures to shorten the QT interval, and direct-current defibrillation when ventricular fibrillation is precipitated. (See also Overview of Arrhythmias.) The long QT interval responsible for torsades de pointes can be congenital or drug-induced.